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Exam Code: C-BCSSS-2502 Dumps

Questions and Answers: 104

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NEW QUESTION: 1


Answer:
Explanation:

Explanation
Box 1:

Box 2:

Box 3:

Box 4:


NEW QUESTION: 2
What does it mean when a node is in pending active state?
A. The node is ready to be activated on the target.
B. The node is ready to be cutover.
C. Inactive data has already been migrated.
D. The migration for a node is preparing to run.
Answer: A

NEW QUESTION: 3
Refer to the exhibit. A network engineer executes the show ipv6 ospf database command and is presented with the output that is shown.
Which flooding scope is referenced in the link-state type?

A. Reserved
B. Link-local
C. Area
D. As (OSPF domain)
Answer: C

NEW QUESTION: 4
Select the class of Anti-diabetic medication that works in the specified organ to prevent hyperglycemia. Select all that applies. Liver (D)
A. DPP4 Inhibitors
B. Glucagon-like peptide-1 receptor agonists
C. Sulfonylureas
D. Biguanide
E. Alpha- Glucosidase Inhibitors
F. Thiazolidinediones
G. SGLT2 inhibitors
Answer: A
Explanation:
Explanation
DPP4 Inhibitors, (D)Glucagon-like peptide-1 receptor agonists, (E)Thiazolidinediones (F)Biguanide Sulfonylureas work in beta cells in the pancreas that are still functioning to enhance insulin secretion. Alpha- Glucosidase Inhibitors stop -glucosidase enzymes in the small intestine and delay digestion and absorption of starch and disaccharides which lowers the levels of glucose after meals. DPP4 blocks the degradation ofGLP-1, GIP, and a variety of other peptides, including brain natriuretic peptide. Glucagon-like peptide-1 receptor agonists work in various organs of the body. Glucagon-like peptide-1 receptor agonists enhance glucose homeostasis through: (i) stimulation of insulin secretion; (ii) inhibition of glucagon secretion; (iii) direct and indirect suppression of endogenous glucose production; (iv) suppression of appetite; (v) enhanced insulin sensitivity secondary to weight loss; (vi) delayed gastric emptying, resulting in decreased postprandial hyperglycaemia. Thiazolidinediones are the only true insulin-sensitising agents, exerting their effects in skeletal and cardiac muscle, liver, and adipose tissue. It ameliorates insulin resistance, decreases visceral fat.
Biguanides work in liver, muscle, adipose tissue via activation of AMP-activated protein kinase (AMPK) reduce hepatic glucose production. SGLT2 inhibitors work in the kidneys to inhibit sodium-glucose transport proteins to reabsorb glucose into the blood from muscle cells; overall this helps to improve insulin release from the beta cells of the pancreas.


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